Journal of IiME Volume 3 Issue 1 www.investinme.org Human enteroviruses and chronic infectious disease (continued) Acknowledgements. Much of our work has been variously supported by the National Institutes of Health, the American Heart Association, the Juvenile Diabetes Research Foundation, and the American Diabetes Association. We also sincerely thank a group of concerned individuals who have lost family due to enteroviral heart disease, for their generous support. References 1. Akerblom, H. K., O. Vaarala, H. Hyoty, J. Ilonen, and M. Knip. 2002. Environmental factors in the etiology of type 1 diabetes. Am J Med Genet 115:18-29. 2. Baboonian, C., M. J. Davies, J. Booth, and W. McKenna. 1997. Coxsackie B viruses and heart disease. Curr Top Microbiol Immunol 223:31-52. 3. Badorff, C., N. Berkely, S. Mehrotra, J. W. Talhouk, R. E. Rhoads, and K. U. Knowlton. 2000. Enteroviral protease 2A directly cleaves dystrophin and is inhibited by a dystrophin-based substrate analogue. J Biol Chem 275:11191-7. 4. Bowles, N. E., P. J. Richardson, E. G. Olsen, and L. C. Archard. 1986. Detection of Coxsackie-B-virus-specific RNA sequences in myocardial biopsy samples from patients with myocarditis and dilated cardiomyopathy. Lancet 1:11201123. 5. Brown, B., M. S. Oberste, K. Maher, and M. Pallansch. 2003. Complete genomic sequencing shows that polioviruses and members of human enterovirus species C are closely related in the noncapsid coding region. J Virol 77:8973-8984. 6. Chapman, N., A. Ramsingh, and S. Tracy. 1997. Genetics of coxsackievirus virulence. Curr Top Microbiol Immunol 223:227-258. 7. Chapman, N. M., and K.-S. Kim. 2008. Persistent coxsackievirus infection: enterovirus persistence in chronic myocarditis and dilated cardiomyopathy. Curr Topics Microbiol Immunol 323:275-292. Invest in ME (Charity Nr. 1114035) 8. Chapman, N. M., K. S. Kim, K. M. Drescher, K. Oka, and S. Tracy. 2008. 5' terminal deletions in the genome of a coxsackievirus B2 strain occurred naturally in human heart. Virology. 9. Chia, J. K., and A. Y. Chia. 2008. Chronic fatigue syndrome is associated with chronic enterovirus infection of the stomach. J Clin Pathol 61:43-8. 10. Cole, C. N., D. Smoler, E. Wimmer, and D. Baltimore. 1971. Defective interfering particles of poliovirus. I. Isolation and physical properties. J Virol 7:478-485. 11. Cunningham, L., N. E. Bowles, R. J. Lane, V. Dubowitz, and L. C. Archard. 1990. Persistence of enteroviral RNA in chronic fatigue syndrome is associated with the abnormal production of equal amounts of positive and negative strands of enteroviral RNA. J Gen Virol 71:13991402. 12. Dalldorf, G. 1955. The coxsackie viruses. Annu Rev Microbiol 9:277-296. 13. Dimmock, N. J. 1991. The biological significance of defective interfering viruses. Rev Med Virol 1:165-176. 14. Drescher, K. M., K. Kono, S. Bopegamage, S. D. Carson, and S. Tracy. 2004. Coxsackievirus B3 infection and type 1 diabetes development in NOD mice: Insulitis determines susceptibility of pancreatic islets to virus infection. Virology 329:381-394. 15. Drescher, K. M., and S. Tracy. 2008. The CVB and etiology of type 1 diabetes. Curr Topics Microbiol Immunol 323:259274. 16. Dutta, F. 2008. Epidemiology of poliomyelitis - options and update. Vaccines 26:5767-5773. 17. Haller, M. J., M. A. Atkinson, and D. Schatz. 2005. Type 1 diabetes mellitus: etiology, presentation, and management. Pediatr Clin North Am 52:1553-78. 18. Kim, K. S., N. M. Chapman, and S. Tracy. 2008. Replication of coxsackievirus B3 in primary cell cultures generates novel viral genome deletions. J Virol 82:2033-7. 19. Kim, K. S., S. Tracy, W. Tapprich, J. Bailey, C. K. Lee, K. Kim, W. H. Barry, and N. M. Page 30/76
31 Publizr Home